Whereas, in 123 patients without significant stenosis, only 1 cardiac event with no severe event was observed. Follow-up information was obtained for occurrence of severe cardiac events (cardiac death, myocardial infarction, and unstable angina) and all cardiac events (additionally including revascularization>90 days after CMRA).ĭuring a median follow-up of 25 months, 10 cardiac events, of which 5 were severe, were observed in 84 patients with significant stenosis. The presence of significant coronary stenosis (≥50% diameter reduction) was visually determined on sliding thin- maximum intensity projection images. We studied 207 patients with suspected coronary artery disease who underwent non-contrast-enhanced free-breathing whole-heart CMRA acquired with a 1.5-T MR system and 32-channel cardiac coils. However, the prognostic value of whole-heart CMRA is unknown. Recent studies demonstrated that the presence of stenosis on coronary computed tomography angiography has a significant prognostic impact on the prediction of cardiac events. This study sought to determine whether whole-heart coronary magnetic resonance angiography (CMRA) can predict cardiac events in patients with suspected coronary artery disease.
When fixed it can be used as a suture anchorage during closure of the ventricular septal defect. When mobile, the tissue must be resected at the time of surgical repair. The precise morphology of the accessory tricuspid valve tissue is of considerable surgical significance. This type created a fixed obstruction of the ventricular septal defect without involving the subaortic left ventricular outflow tract. The "fixed" variety was attached to the edges of the defect by short chordae which reduced considerably its movements. The "mobile" variety was tethered by long chordae tendineae which permitted a wide excursion of the leaflet through the ventricular septal defect into the left ventricular outflow tract where it represented a potential cause of obstruction.
Depending on the morphology, the accessory tissue was classified into "mobile" or "fixed" types. This structure caused partial or almost complete obstruction of the ventricular septal defect.
Among 61 heart specimens of tetralogy of Fallot with or without pulmonary atresia, four presented with an accessory tricuspid valve leaflet.